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Chapter 1 Chairman's beginning feedback (pages 1–4): Professor R. E. Tunbridge
Chapter 2 Diabetes in a Village group (pages 5–24): Joan B. Walker
Chapter three Spontaneous Diabetes Mellitus within the chinese language Hamster, Cricetulus griseus. IV. Genetic features (pages 25–48): George Yergaman
Chapter four The Endocrine Pancreas in Human and Experimental Diabetes (pages 49–74): Robertson F. Ogilvie
Chapter five Pancreatic Beta mobile (pages 75–88): Paul E. Lacy
Chapter 6 Panel dialogue on Biosynthesis and Secretion of Insulin: Biosynthesis of Insulin (pages 89–94): ok. W. Taylor
Chapter 7 Insulin Biosynthesis and Glucose Metabolism in Islet arrangements (pages 95–98): Albert E. Renold
Chapter eight Secretion of Insulin precipitated by way of Leucine in fit matters (pages 99–106): Stefan S. Fajans, John C. Floyd, Jr. Ralph F. Knopf and Jerome W. Conn
Chapter nine expense of liberate of Insulin in vitro (pages 107–121): P. J. Randle
Chapter 10 Immunological reviews with Homologous and Heterologous Pancreatic Insulin within the Cow (pages 122–139): Albert E. Renold, Stuart Soeldner and Jurgen Steinke
Chapter eleven Plasma Insulin in Diabetes (pages 140–155): C. N. Hales
Chapter 12 Insulin Antagonism: primary concerns (pages 156–191): David M. Kipnis and Martin F. Stein
Chapter thirteen The Glucose Fatty Acid Cycle and Diabetes Mellitus (pages 192–216): P. J. Randle, P. B. Garland, C. N. Hales and E. A. Newsholme
Chapter 14 Synalbumin Insulin Antagonism and Diabetes (pages 217–239): J. Vallance?Owen
Chapter 15 Pituitary Polypeptide Antagonists of Insulin (pages 240–249): J. Bornstein, Deirdre Hyde and ok. J. Catt
Chapter sixteen Peripheral motion of Insulin Diabetes Mellitus and its Relation to the Aetiology of Diabetes Mellitus (pages 250–281): W. J. H. Butterfield
Chapter 17 The Biochemistry of Blood Vessels (pages 282–300): Helen Muir
Chapter 18 The Glomerular Lesion in Idiopathic and Secondary Diabetes (pages 301–314): M. ok. Macdonald and J. T. Ireland
Chapter 19 Pathophysiology of Microangiopathy in Diabetes Mellitus (pages 315–344): Searle B. Rees, Rafael A. Camerini?Davalos, James B. Caulfield, Oscar Lozano?Castaneda, Alfredo Cervantes?Amezcus, Jan Taton, Daniel Pometta, Jorge P. Krauthammer and Alexander Marble
Chapter 20 Panel dialogue at the position of the Pituitary in Human Diabetes and its problems within the Aetiology of Diabetes Mellitus: The Anterior Pituitary (pages 345–347): J. Bornsteiy
Chapter 21 The Roe of the Pituitary in Human Diabetes (pages 348–353): R. Luft
Chapter 22 The influence of Yttrium?90 Pituitary Implantation on Diabetic Retinopathy (pages 354–390): G. F. Joplin
Chapter 23 ultimate comments (pages 391–392):
Read Online or Download Ciba Foundation Symposium - Aetiology of Diabetes Mellitus and its Complications (Colloquia on Endocrinology), Volume 15 PDF
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Additional resources for Ciba Foundation Symposium - Aetiology of Diabetes Mellitus and its Complications (Colloquia on Endocrinology), Volume 15
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W. Clin. , 6, 251 [abstract]). These were all non-prcgnant individuals. It has already been pointed out that in pregnancy there is both an increased glomerular filtration rate and a decreased maximum tubular reabsorptive capacity for glucose. W e have no information whether renal glycosuria during pregnancy might have similar implications in suggesting a predisposition to future diabetes. Thinbridge: It is extremely rare, however carefully the history is taken, to get hypoglycaemia in a person who is a true diabetic, but one often gets diabetic curves in people who have spontaneous hypoglycaemia.
73, 166), who substantiated the earlier findings that the lesion is definitely the result of the metabolic sequences rather than a response to acute ageing and such. Rees: Kuwabara and Cogan (personal communication) have demonstrated mural cell degeneration in Dr. Yerganian’s diabetic Chinese hamsters. Professor Ashton is quite right in pointing out that the retinopathy described by Hausler in the diabetic Chinese hamster is not the exact equivalent of human diabetic retinopathy, but it is the best experimental model available for study at the present time.
For example, some animals had completely lost the internal structure of one kidney and remnants of tubules floated in an otherwise fully distended and "bladder-like" organ. This extent 28 G . YERGANIAN of degencration was not witnessed in later cases, as reported by Meier and Yerganian (1959)and Lawe (1962),where bilateral distention of the pelves and calyces was either mild or severe and renal parenchyma appeared normal. Histological preparations disclosed that periodic acid-Schiffpositive matcrial within capillary tufts completely obliterated glomerular structures, in addition to accumulating in the bascment membrane.